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Juan Reyes

Principal Research Engineer

Publications

2022

Carla Azevedo, Gabriel Teku, Yuriy Pomeshchik, Juan F. Reyes, Margarita Chumarina, Kaspar Russ, Ekaterina Savchenko, Anna Hammarberg, Nuno Jorge Lamas, Anna Collin, Gunnar K. Gouras, Oxana Klementieva, Martin Hallbeck, Ricardo Taipa, Mauno Vihinen, Laurent Roybon (2022) Parkinsons disease and multiple system atrophy patient iPSC-derived oligodendrocytes exhibit alpha-synuclein-induced changes in maturation and immune reactive properties Proceedings of the National Academy of Sciences of the United States of America, Vol. 119, Article e2111405119 Continue to DOI
Serge Moudio, Fredrik Rodin, Nazira Albargothy, Urban Karlsson, Juan Reyes, Martin Hallbeck (2022) Exposure of alpha-Synuclein Aggregates to Organotypic Slice Cultures Recapitulates Key Molecular Features of Parkinsons Disease Frontiers in Neurology, Vol. 13, Article 826102 Continue to DOI

2021

Juan Reyes, Sara Ekmark-Lewen, Marina Perdiki Grigoriadi, Therése Klingstedt, Alana Hoffmann, Emilia Wiechec, Per Nilsson, Peter Nilsson, Irina Alafuzoff, Martin Ingelsson, Martin Hallbeck (2021) Accumulation of alpha-synuclein within the liver, potential role in the clearance of brain pathology associated with Parkinsons disease Acta neuropathologica communications, Vol. 9, Article 46 Continue to DOI

2019

Juan Reyes, Christopher Sackmann, Alana Hoffmann, Per Svenningsson, Juergen Winkler, Martin Ingelsson, Martin Hallbeck (2019) Binding of α-synuclein oligomers to Cx32 facilitates protein uptake and transfer in neurons and oligodendrocytes Acta Neuropathologica, Vol. 138, p. 23-47 Continue to DOI

2018

Anna Ansell - Schultz, Juan Reyes, My Samuelsson, Martin Hallbeck (2018) Reduced retromer function results in the accumulation of amyloid-beta oligomers Molecular and Cellular Neuroscience, Vol. 93, p. 18-26 Continue to DOI

News

3D illustration brain

A newly discovered mechanism reveals how Parkinson’s disease can spread

Tiny channels between nerve cells are involved in a newly discovered mechanism of how Parkinson’s disease can spread throughout the brain. Harmful protein aggregates can “hitch a lift” with channel-forming proteins, and spread to healthy cells.

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