Fotografi av Sarah Lindström

Sarah Lindström

Förste forskningsingenjör

Forskning

Publikationer

2023

Teresa Mínguez‐Viñas, Varsha Prakash, Kaiqian Wang, Sarah Lindström, Serena Pozzi, Stuart A. Scott, Elizabeth Spiteri, David A. Stevenson, Euan A. Ashley, Cecilia Gunnarsson, Antonios Pantazis (2023) Two epilepsy‐associated variants in KCNA2 (KV1.2) at position H310 oppositely affect channel functional expression Journal of Physiology, Vol. 601, s. 5367-5389 (Artikel i tidskrift) Vidare till DOI

2022

Michelle Nilsson, Sarah H Lindström, Maki Kaneko, Kaiqian Wang, Teresa Minguez-Viñas, Marina Angelini, Federica Steccanella, Deborah Holder, Michela Ottolia, Riccardo Olcese, Antonios Pantazis (2022) An epilepsy-associated KV1.2 charge-transfer-center mutation impairs KV1.2 and KV1.4 trafficking Proceedings of the National Academy of Sciences of the United States of America, Vol. 119 (Artikel i tidskrift) Vidare till DOI

2020

Antonios Pantazis, Maki Kaneko, Marina Angelini, Federica Steccanella, Annie M. Westerlund, Sarah Lindström, Michelle Nilsson, Lucie Delemotte, Sulagna C. Saitta, Riccardo Olcese (2020) Tracking the motion of the K(V)1.2 voltage sensor reveals the molecular perturbations caused by ade novomutation in a case of epilepsy Journal of Physiology, Vol. 598, s. 5245-5269 (Artikel i tidskrift) Vidare till DOI
Sarah Lindström, Sofie Sundberg, Max Larsson, Fredrik K. Andersson, Jonas Broman, Björn Granseth (2020) VGluT1 Deficiency Impairs Visual Attention and Reduces the Dynamic Range of Short-Term Plasticity at Corticothalamic Synapses Cerebral Cortex, Vol. 30, s. 1813-1829 (Artikel i tidskrift) Vidare till DOI

2018

Anna Klawonn, Daniel Wilhelms, Sarah Lindström, Anand Kumar Singh, Maarit Jaarola, Jurgen Wess, Michael Fritz, David Engblom (2018) Muscarinic M4 Receptors on Cholinergic and Dopamine D1 Receptor-Expressing Neurons Have Opposing Functionality for Positive Reinforcement and Influence Impulsivity Frontiers in Molecular Neuroscience, Vol. 11, Artikel 139 (Artikel i tidskrift) Vidare till DOI

Organisation

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